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We found this paradox to be explained by GLP-1 delaying gastric emptying and the rate of carbohydrate absorption after a meal, biological actions that dominant direct effects on insulin secretion at the B-cell, both in both normal-weight individuals and in non-diabetic individuals with obesity [6, 10] (Fig
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The peak occurred at 3 h pi , followed by a continuous decrease until 9 h pi and a subsequent return to the initial level
Rapid phosphorylation of insulin signaling molecules in response to glucose intake was observed, in contrast to the more gradual changes in gene expression
McDonnell, E
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