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The treatment with FMN reduced programmed cell death in renal tubular cells and prevented mitochondrial fragmentation, concurrently restoring levels of key proteins involved in mitochondrial dynamics (Drp1, Fis1, MFN2) as well as apoptotic regulators (Bax, Bcl-2, Caspase-3)
From Week 6 onward, the p-values showed no significant difference between groups
Glutathione deficiency increases oxidative stress
doi: 10.1146/annurev.nutr.24.012003.132120 Summary Keywords selenium, selenoprotein, thyroid disease, oxidative stress, iodine Citation Wang F, Li C, Li S, Cui L, Zhao J and Liao L (2023) Selenium and thyroid diseases
By contrast, a case report described a man who survived after ingesting as much as 60 grams all at once
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