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Meanwhile, the ectopic deposition of in the ovaries and liver directly impairs oocyte mitochondrial function and energy metabolism, resulting in the so-called lipotoxic oocyte phenotype (64, 65)

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In vitro studies have demonstrated that BPC-157 can activate signaling pathways that promote fibroblast survival and growth, even under conditions of oxidative stress or hypoxia that often characterize the microenvironment of an injured tendon

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