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BMC Vet Res 13:112 Al-Zubaidy A, Khalil A (2022) Gastroprotective effect of Capparis spinosa on indomethacin-induced gastric ulcer in rats
Without adequate glutathione to neutralize ROS, superoxide radicals collide with and damage the mitochondria's inner membrane (specifically a lipid called cardiolipin) and the mitochondrial DNA itself
This prevents its degradation in the digestive tract
The increase in ROS production caused by hyperglycemia occurs through the following ways: activation of the protein kinase C (PKC) pathway via diacylglycerol (DAG), increased hexosamine pathway flux, increased production of advanced glycation-end product, and increased flux in the polyol pathway (32, 33)
