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Mechanisms of estradiol-induced insulin secretion by the G protein-coupled estrogen receptor GPR30/GPER in pancreatic -cells

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The subjects were equally block-randomized into three groups: GSSG (250 mg/d), GSH (250 mg/d), or placebo

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Second, its relatively small size and amphiphilic character allow partial blood-brain barrier transit, but enzymatic degradation in plasma competes with CNS entry at every moment after injection

Semaglutide disrupts at least three of those conditions