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The molecular events leading to activation or silencing of glucose-sensitive neurons are surprisingly similar to those of the endocrine pancreas and include neuronal uptake of glucose via GLUT2, conversion to pyruvate (and finally ATP) via glucokinase, and modulation of neuronal firing mediated by changes in K ATP channel activity, cell depolarisation and Ca 2+ influx
For many patients, treatment options may include FDA-approved medications such as semaglutide, which works to regulate appetite and support weight loss when combined with other healthy habits
Kiernan et al
Pancreas 44 , 799804 (2015)
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