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By binding to IL-17A and preventing it from interacting with its receptor, Taltz interrupts the inflammatory cascade that causes skin plaques, joint inflammation, and spinal rigidity (Langley et al., New England Journal of Medicine , 2014)
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J., Tan, D
These results suggest that although the X(GD_L) proteases, including X(GD), are less catalytically active than wild-type BoNT/X protease and X(PC), they are more selective
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