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In order to limit the release of payloads before internalization, preventing or minimizing degradation outside the target cell, the proteome of the lysosome became a logical place to search for enzymes capable of ADC degradation and present in high concentration

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reported that [Ser (2) exendin (1-9)], a GLP-1R agonist, significantly attenuated kainic acid-induced apoptosis in the CA3 region of the hippocampus (117)

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