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Researchers note that this decline coincides with the noticeable decrease in regenerative capacity of an organism. This correlation suggests that decreased natural GHK levels may play a role in slower wound healing observed in older adults

GHK-Cu operates through natural signaling pathways rather than competing for metabolic enzymes or receptor sites used by most pharmaceutical compounds

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The result is increased nitric oxide production, which dilates blood vessels and promotes the formation of entirely new vascular networks at injury sites

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