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The answer is yes
Importantly, Epitalon treatment significantly reduced H2AX expression, suggesting that the peptide alleviates aging-induced genomic stress [2]
The present study demonstrated a concentration-dependent vasodilation effect of BPC 157 in isolated rat aorta
But here's the problem
The proposed mechanism for the BPC-157 component is pathway-level throughout: reported sensitivity of effects to manipulation of the nitric oxide system, and reported changes in growth-factor and adhesion-signaling readouts
This has implications for synthesis, stability and analytical control
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