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Antioxidants are the bodys primary defense against this damage
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It activates beta-3 adrenergic receptors located in fat tissue, which triggers lipolysis (fat breakdown) without sending any signals to the brains hunger centers in the hypothalamus
This paper demonstrates the importance of dysregulation of stress granule dynamics and its interaction with nucleocytoplasmic transport as a pathophysiological mechanism contributing to motor neuron injury in models of ALS
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