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Deficiency of 5-methyladenosine phosphorylase (MTAP), a key enzyme in polyamine metabolism in tumors, can accelerate the accumulation of 5-deoxy-5methylthioadenosine (MTA) in the TME, ultimately reducing the maturation of dendritic cells and the activation of T cells

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The authors would like to express their sincere gratitude to the staff of Kliniskt Forskningscentrum, Biobanken Norr, and Klinisk Kemi at Ume University Hospital for providing excellent research conditions, with special thanks to Camilla Ring, Inger Arnesj, Ingegerd Valinger, Kerstin Enquist Olsson, Gunilla Backman, and Lena Nordstrm

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